Trophic changes can be caused by various diseases associated with cardiovascular dysfunction. Ulcers are most often observed on the lower extremities, but other parts of the body are not immune to these pathologies.
In approximately one in six clinical cases, a combination of several risk factors is observed, contributing to the development of deep defects in the skin and subcutaneous structures. The most common combination is the simultaneous presence of venous and arterial pathologies, as well as a specific complication of diabetes known as angiopathy. This multi-component nature of the lesion significantly complicates diagnosis and treatment, since each component can exacerbate the manifestations of another.
The formation of purulent skin lesions is caused by a number of serious pathological conditions:
When identifying and eliminating the causes of trophic changes, it is important to use a comprehensive approach.
Statistics are relentless: the lower extremities are the primary location for the majority of trophic ulcers. This is explained by the anatomy of the human body.
Firstly, our legs experience enormous pressure from our body weight every day. They are constantly subjected to stress when walking, standing, and playing sports. This creates conditions for prolonged compression of superficial and deep blood vessels, impairing circulation and causing a deficiency of oxygen and nutrients in cells.
Secondly, the structure of the venous system in the legs deserves special attention. It is designed in such a way that blood is forced to overcome gravity, rising upward. A third important factor is that the skin and soft tissues of the legs are thinner and less dense than those of the torso and arms. This makes the legs particularly vulnerable to mechanical injury, irritation, and infection.
The development of the pathology begins with a subtle sign—the appearance of a small patch of grayish-blue skin. Soon, the person begins to experience discomfort in the affected area: a feeling of fullness, an unpleasant tingling sensation, discomfort, and irritation, gradually developing into pain.
Then the skin noticeably thickens, loses elasticity and smoothness, and becomes denser than usual. The next stage is marked by the formation of a moist, erosive surface that rapidly expands in diameter. Upon detecting the first alarming symptoms, it is extremely important to immediately consult a phlebologist or a surgeon specializing in vascular diseases.
The progression of a trophic ulcer proceeds sequentially through four key phases:
The lack of timely medical intervention dramatically increases the risk of the disease progressing to severe forms.
Many people try to combat the condition by using disinfectant solutions such as brilliant green, antibacterial ointments, and drying compounds, but this negatively impacts the healing process of trophic ulcers. Prolonged treatment with such agents can significantly slow tissue repair, provoke allergic reactions, and develop drug resistance in infectious agents.
If dangerous bacterial strains such as Staphylococcus aureus or Pseudomonas aeruginosa are present, broad-spectrum antibiotics are advisable, although they should only be used when other methods have proven ineffective.
The most effective method of conservative treatment for venous trophic ulcers is the use of specialized elastic bandages or special medical stockings. In addition, medications are prescribed to improve vein tone and strengthen the capillary system.
Primary surgical debridement—a radical operation to remove nonviable tissue—is recognized as the optimal method for cleaning ulcerative lesions. The procedure is performed using modern technologies: laser surgery, ultrasound equipment, or hydrosurgical devices under local anesthesia.
Once the surgery is completed, measures are taken to stimulate the formation of new, healthy cells. If complete wound closure does not occur after effective cleansing and active growth of new tissue, the next step is skin flap transplantation.
The main goal of treating trophic leg ulcers with varicose veins is to stop the abnormal backflow of blood into the superficial veins. This is achieved using modern technologies, such as laser coagulation. A key feature of this method is its minimal trauma: the procedure is performed on an outpatient basis, under local anesthesia, without incisions. The laser beam heats the inner lining of the vein, causing its walls to adhere together and form a fibrous band. This stops the abnormal blood flow characteristic of varicose veins, improving the patient's overall well-being.
Foam sclerotherapy is an alternative to surgical treatment for venous trophic ulcers. A series of sessions stops localized blood flow and restores normal circulation in the leg, regardless of the stage of trophic changes. This method is especially recommended for elderly patients with severe skin damage due to advanced varicose veins.
Another innovative approach to treating trophic ulcers is vacuum therapy, which creates negative pressure directly within the wound cavity. A special device gently draws out fluid and bacteria, improves blood flow, and stimulates new tissue formation. The technique involves placing a sterile dressing with a soft drain over the wound, connected to a pump that creates a vacuum.
The K+31 Clinic specializes in the diagnosis and comprehensive treatment of trophic ulcers, offering patients effective solutions and a high standard of medical care.
Our advantages:
We believe that a sound treatment strategy for trophic ulcers should include an accurate diagnosis, comprehensive patient preparation, and ongoing monitoring of the effectiveness of the chosen methods. This is precisely the approach we employ in our clinics.
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Definition and classification of trophic ulcers
A trophic ulcer is a chronic injury to the skin and underlying tissues that is difficult to heal. The primary cause of chronic, non-healing wounds is impaired blood supply and neurotrophic regulation of the affected areas.
Such pathologies typically arise as a result of chronic diseases such as varicose veins, diabetic vascular disease, and arterial disorders. Among the most common patient complaints are pain, hyperemia, and swelling in the affected area. The severity of clinical manifestations varies significantly depending on the stage of the pathological process.
Regeneration in such cases is complicated by profound metabolic disorders in the body, which impede normal tissue repair. Complex therapy is aimed at eliminating the underlying cause of ulcer formation, activating tissue repair processes, and preventing possible complications.
According to the cause of their occurrence, trophic ulcers are divided into several types:
Each form of pathology requires individual approach to therapy, for example, in the treatment of venous trophic ulcers, the basis is the elimination of blood stagnation.